片语

Human ADMA人不对称二甲基精氨酸

ADMA Group先进材料集团

p-ADMA(Padma)人名；(尼)帕德马

ADMA Forum澳大利亚直销协会论坛

ADMA AND EVE亚当与夏娃

ADMA BAILEN身边的人

adma concentrationadma浓度

adma atil帕德玛

Adma Freeman精通ASP

英汉例句

• Conclusion taurine protects the endothelium against damages induced by LDL, and the protective effect of taurine on endothelium is related to the reduction of ADMA concentration in rats.

  结论牛磺酸对LDL诱导的血管内皮细胞损伤有保护作用，其保护作用与降低adma浓度有关。

• The plasma ADMA levels are elevated in EH patients.

  ②高血压病患者血浆ADMA的水平升高；

• This article reviews the effects of ADMA on endothelial function and its mechanism and the research into related therapeutic procedures.

  现就adma对内皮功能的影响和机制以及相关的治疗研究现状进行综述。

• Conclusions: Plasma ADMA levels predict fatal and non-fatal cardiovascular events in patients with type 1 nephropathy.

  结论：在1型糖尿病肾病患者中，血adma的水平对致命和非致命的心血管事件有预测作用。

• CONCLUSION: Endothelial dysfunction induced by OFR is associated with the increase in ADMA concentration and reduction of DDAH activity, but not DDAH expression.

  结论：OF R培养下，内皮损伤adma的增加与DDAH的活性减弱有关，而与DDAH的表达无关。

• These results suggest that in the older group of rats, the elevated level of ADMA may be a contributor to vascular endothelial dysfunction.

  提示老龄大鼠血中二甲基精氨酸浓度升高可能是促进血管内皮功能不全的重要原因。

• In a second model, ADMA proved to be a significant independent risk marker of CHD, along with hypercholesterolemia, hypertension, diabetes, and smoking.

  在第二个模型中，ADMA被证明是冠心病显着的、独立的标志物，伴随高胆固醇血癥、高血压、糖尿病和吸烟。

• Fenofibrate reversed the effect of ADMA on the expression of MMP-9 and promoted the synthesis of no.

  非诺贝特可以抑制adma诱导人脐静脉内皮细胞所致MMP - 9的表达增加并促进NO的合成。

• In the overall population, ADMA plasma levels correlated with body-mass index, creatinine levels, and plasma triglyceride levels, but not with age, the results indicate.

  在全部人群中，ADMA血浆水平与体重指数、肌氨酸酐水平和血浆甘油三酸酯水平相关，但与年龄、结果显示不相关。

• Furthermore, increased ADMA levels tended to contribute to increased risk of progressive diabetic kidney disease.

  并且ADMA水平升高促进糖尿病肾病发展。

• The ADMA plasma level also tended to increase with an increasing number of classic cardiovascular risk factors, the researchers note.

  研究者还注意到：adma血浆水平倾向于随着传统的心血管风险因子种类的增加而增加。

• CONCLUSIONS: OFR induces endothelial dysfunction through increasing ADMA concentration, while captopril relieves endothelial dysfunction induced by ox-LDL through decreasing ADMA concentration.

  结论：OFR通过增加ADMA导致内皮功能紊乱，卡托普利则能通过减少ADMA减轻OFR诱导的内皮细胞代谢功能障碍。