conclusion:laao of king cobra venom can induce the apoptosis of nacc cells.
结论:眼镜王蛇毒laao能诱导nacc细胞发生凋亡。
methods a model of neuronal injury was established on sh-sy5y cells through activation of complement by cobra venom factor, an alternative pathway-specific activator.
方法采用眼镜蛇毒因子特异激活血清补体,诱导人神经母细胞瘤细胞sh-sy5y损伤。
aim: to explore the immunoreactivity and neutralization against king cobra venom of anti-king cobra venom igy.
目的:探讨抗眼镜王蛇毒卵黄抗体的免疫活性及其在体内外对眼镜王蛇毒的中和作用。
aim to investigate the protective effect of nerve growth factor (ngf) from the chinese cobra venom on spinal neurons after sciatic nerve lesion.
目的探讨中华眼镜蛇毒神经生长因子对受损神经元的保护作用。
objective to investigate the effect of purified cobra venom factor (cvf) in rats and the efficacy in preventing hyperacute rejection (har) after discordant xenotransplantation.
目的探讨眼睛蛇毒因子(cvf)对血清总补体活性及对豚鼠到大鼠异种心脏移植超急性排斥反应的影响。
objective to investigate the effects of chinese cobra venom factor(ccvf) and dan-shen root on small intestine xenograft survival.
目的探讨中华眼镜蛇毒因子、丹参对异种小肠移植存活的作用。
the results show that naja naja atra cobra venom injection is safe for therapeutic use.
实验结果提示,蛇毒注射液用药是安全的。
to prepare plga microspheres of cobra venom cytotoxin and study its characterization and characteristics of in vitro release; 3.
制备眼镜蛇毒细胞毒素plga微球,研究其表征及体外释药性质;3。
in guinea pig to rat heart xenotransplantation model, cobra venom factor is administrated to avoid hyperacute rejection, and thus delayed xenograft rejection model is made.
在豚鼠—大鼠异种心脏移植中,给以眼镜蛇毒因子以避免发生超急性排斥反应,制作延迟性排斥反应模型。
cobra venom factor (cvf) is a nontoxic glycoprotein present in cobra venom with strong complement-activating activity through the alternative pathway.
眼镜蛇毒因子(cvf)是一种具有补体激活活性的糖蛋白。
guangxi cobra venom can inhibit proliferation and induce apoptosis of nacc cell line in vitro.
结论眼镜蛇毒能抑制nacc细胞增殖并诱导其凋亡。
conclusion: the cobra venom cytotoxin had a high cytotoxic activity on cultured human cancer cell lines.
结论:眼镜蛇毒细胞毒素对体外培养的人癌细胞有很强的杀伤作用。
objective:to separate and purify the hypotensive factor(hf) from cobra venom of guangxi and study its biological activity, hypotensive effect, inhibition of cardiomyocyte hypertrophy and cytotoxicity.