object: to investigate the effect of aiweixin on the heart function, arrhythmia, ldh, formazan content and myocardial apoptosis of isolated rat heart during ischemia and reperfusion.
目的:观察爱维心对离体缺血再灌注过程心脏心功能、心律失常、心肌酶、心肌细胞活性及其心肌细胞凋亡的影响,并探讨其可能的作用机制。
conclusions ip in isolated rat heart can induce infarct size restraint and cell kinase leakage reduction.
结论ip可缩小心肌梗死面积,减少细胞酶渗漏;
bioactivity test on isolated rat heart failure model suggested 1 could increase coronary blood flow and myocardial con- tractive power compared with positive control ouabain.
离体大鼠心衰试验表明,与对照药哇巴因比较, 化合物1能增加冠脉血流量和增强心肌收缩力。
objective to investigate the role of pi3k/akt signaling pathway in the protection of isolated rat heart from ischemia-reperfusion(i/r)injury by propofol.
目的探讨pi3k/akt信号传导通路在异丙酚减轻离体大鼠心脏缺血再灌注损伤中的作用。
an isolated rat heart working model was established.
建立离体心工作模型;