objective to investigate the mechanism of reversion of myocardial interstitial fibrosis in diabetic cardiomyopathy (dcm) by valsartan.
目的探讨缬沙坦逆转糖尿病心肌病(dcm)心肌间质纤维化的作用机制。
objective to assess left ventricular myocardial interstitial fibrosis and effects of valsartan in diabetic cardiomyopathy(dcm) rats by integrated backscatter (ibs) technique.
目的应用超声背向散射积分(ibs)技术评价糖尿病心肌病(dcm)大鼠左室心肌间质纤维化及缬沙坦的治疗效果。
electromicroscopy pointed to myocardial interstitial fibrosis and mc infiltration, whose presence in cirrhotic myocardium may have played a role in the pathogenesis of cirrhotic cardiomyopathy.
结果表明肝硬化存在心肌间质纤维化和肥大细胞浸润,可能参与肝硬化心肌病的发生。
conculusion simvastatin could attenuate the development of myocardial interstitial fibrosis in non-infarction zone to improve myocardial interstitial remodeling in rats after mi.
结论辛伐他汀能减少梗死边缘区胶原沉积,改善心肌梗死后心肌间质重构。
conclusion fluvastatin could inhibit myocardial interstitial fibrosis and improve the cardiac function of diabetic rats, which might be related to the down-regulating of the ctgf expression.
结论氟伐他汀可逆转糖尿病大鼠心肌间质纤维化并改善心功能,这种作用可能与其抑制ctgf的表达有关。