Conclusion: increase of ET 1 is an important factor leading to ischemic brain damage after SAH. GBE exerts its protective effect by antagonizing pathological increase of ET 1.
Method: GBE sustained-release tablets were prepared by direct compression method using sodium carboxymethylcellulose CMC-Na and hydroxypropyl methylcellulose HPMC as matrix excipients.
方法:以溶胀性材料羧甲基纤维素钠和羟丙甲基纤维素为骨架材料,直接粉末压片制备银杏提取物缓释片。
CONCLUSION: GBE may relieve cerebral ischemic damage after SAH.
结论:GBE可减轻SAH后缺血性脑损伤。
Conclusion: GBE can delay demyelination process in EAE mice by inhibiting microglial activation, suggesting that GBE has potential to treat multiple sclerosis in future.
AIM: To study the protective effects of CO GBE on multi infarction dementia (MID) rat model.
目的:探讨复方银杏滴丸(COGBE)对多发性脑梗死癡呆(MID)大鼠的保护作用。
RESULTS: 1 Compared with diabetes group, retinal vessel endothelium and ganglion cell apoptosis decrease much less in GBE treatment group.
结果:GBE治疗组视网膜内皮细胞、神经节细胞凋亡明显较糖尿病组减轻。
EEG: The EEG of normal saline control group and GBE control group was normal.
生理盐水对照组和GBE单独给药对照组大鼠eeg正常。
Objective To explore the effects of Ginkgo Biloba Extract (GBE) on the PTZ-induced seizures and the protection of GBE on the brain injury by epileptic seizures.
目的探讨银杏叶提取物(GBE)对癫痫发作的影响及对癫痫发作后脑损伤的保护作用。
Objective To investigate the change of ocular hemodynamics in patients with chronic glaucoma who received GBE after trabeculect o my with color doppler imaging (CDI).