CLL requiring treatment develops in subjects with CLL-phenotype MBL and with lymphocytosis at the rate of 1.1% per year.
在有CLL-表型的单克隆B淋巴细胞增多和以每年1.1%速度增加的淋巴细胞增多癥患者需要CLL的治疗。
Recently, more and more data indicate that MBL insufficiency is closely associated with recurrent infection of a wide range pathogen.
近年来,越来越多的研究表明,各种病原体的反复感染与MBL缺损密切相关。
Conclusion Among the PA strains isolated from clinic, both MBL producing strains and non-MBL ones were multiple antimicrobial resistance.
结论临床感染分离的PA、产酶菌株与非产酶菌株均呈多重耐药性。
Conclusions - These results are the first to show that MBL is abundantly present and locally produced during early atherogenesis.
结论:这些结果首次提示MBL在动脉粥样硬化早期局域性大量表达。
MBL was present and was produced in 10-week-old lesions, whereas deposition and gene expression were minimal after 18 weeks of high-fat feeding and absent in healthy vasculature.
MBL在8周龄小鼠动脉粥样硬化损伤部位开始产生出现,高脂饮食18周后组织蛋白沉积和基因表达比较轻微,而在健康血管中没有相应表达。
The purified recombinant product could react with the antibody against the recombinant human MBL protein in the indirect ELISA.
ELISA检测证实,纯化的蛋白能与鼠抗重组人MBL蛋白抗体结合。
Objective: To discuss mannose binding lectin (MBL) gene polymorphisms at 54 locus of Han population in Ningxia for further research on relationship of MBL gene mutation and HBV infection.
目的:探讨宁夏汉族人群中甘露糖结合凝集素(MBL)基因54位点多态性,为进一步研究MBL基因突变与乙型肝炎间的关联性提供依据。
However, the relationship between structure and functions of MBL and the mechanisms by which MBL structure gene mutations cause opsonic deficiency are to be clarified.
目前,MBL的结构-功能关系未明,MBL基因突变引起调理吞噬缺损的机制不清,这些均需进一步研究予以阐明。
Local MBL expression, by myeloid cells, is shown to critically control development of atherosclerotic lesions.
髓系细胞表达的MBL在动脉粥样硬化损伤进展中起着临界控制的作用。
Methods:The method for MBL point mutation detection(PCR-RFLP) was Established with self-designed primers according to MBL genomic sequence;
方法:根据MBL基因序列设计引物建立MBL基因点突变检测方法即PCR -RFLP ;
The MBL is the number of U. S. farms recognized quality brand, making it richer and more extensive product.
而MBL是美国许多鸡场认可的优质品牌,从而使其产品更丰富和广泛。
Subsequently, analysis of MBL deposition and gene expression in advanced human atherosclerotic lesions revealed the presence of MBL protein in ruptured but not stable atherosclerotic lesions.
随后发现人进展期动脉粥样硬化损伤斑块中,破裂斑块中有MBL蛋白沉积和基因表达,而稳定性斑块中没有相应表现。