objective to investigate the effect of fibrin deposition on endothelial cell function in pulmonary tissue of rats with different ages.
目的探讨纤维蛋白沉积对不同鼠龄大鼠肺组织血管内皮细胞功能的影响。
conclusions aging could accelerate fibrin deposition in pulmonary tissue of lps-induced rat model of acute lung injury, and promote the injury of endothelial cell function caused by fibrin deposition.
结论增龄能够促进脂多糖诱导的急性肺损伤大鼠肺组织纤维蛋白沉积,增强纤维蛋白的内皮细胞损伤作用。
recent data indicated that aging accelerated glomerular fibrin deposition induced by lipopolysaccharide (lps) in mice, and that fibrin could directly activate endothelial cell in vitro.
研究表明,衰老能够促进脂多糖(lps)诱导的小鼠肾小球纤维蛋白沉积。
background. intraglomerular fibrin deposition has been observed in most types of human glomerulonephritis and associated with glomerular injury.