英汉例句

• yet, while we are beginning to understand the cellular mechanisms underlying synaptic changes, it is important to consider the functional implications of synaptic plasticity in the intact brain.

  当开始了解这些突触改变的细胞生物学机制的时候，也应该考虑这种突触可塑性在完整大脑中的功能意义。

• effect of modafinil on cognition and synaptic plasticity after rem sleep deprivation in rats;

  目的探讨大鼠脑梗死后树突可塑性的物质基础和机制及针刺的影响。

• the paper is a summary about the researches of motor learning in the nerve routes and synaptic plasticity and discusses their practical sense as well.

  本文综述这方面的研究成果并讨论了它们的实践指导意义。

• synaptic plasticity is important for the remodeling of neural circuits during brain development and is implicated in higher brain functions such as learning and memory.

  突触可塑性对于脑发育过程中的神经环路重构以及学习记忆等脑的高级功能是非常重要的。

• changes in synaptic plasticity in dopamine neurons of the ventral tegmental area (vta) may contribute to circuit modification induced by many drugs of abuse, including cocaine.

  腹侧被盖区多巴胺神经元的突触可塑性的改变可能在许多药物滥用（包括可卡因滥用）所导致的回路修饰中起着一定作用。

• we explored the effects of prenatal stress on hippocampal synaptic plasticity and behavior of the offspring by testing ltp and ltd in hippocampal ca1 area and learning and memory in morris water maze.

  通过对出生前有慢性应激经历子代海马ca1区ltp和ltd诱导性及学习记忆和行为反应的测定，探讨出生前慢性应激对子代海马ca1区突触可塑性及学习记忆行为的影响。

• postsynaptic dense area and the close relationship between synaptic plasticity are essential for the maintenance of synaptic function properly.

  突触后致密区与突触可塑性的关系十分密切，对于维持突触发挥正常功能至关重要。

• we studied the effects of pbde209 on synaptic plasticity in the dg in vivo, and discussed different effects of pbde209 in the different developmental periods.

  并且在不同发育期其对突触可塑性的损害不同，其中哺乳期是大鼠对pbde209最敏感的时期。

• c and synaptic plasticity may exist in the respiratory neurons of b? t. c.

  有关的神经元突触在功能上具有一定的可塑性。

• synaptic plasticity often affects the shape of synaptic structures.

  突触可塑性常常影响突触结构的形状。

• the scientists point out that alcohol isn「t causing blackouts by killing neurons. instead, the steroids interfere with synaptic plasticity to impair ltp and memory formation.

  研究人员指出，酒精并不是通过杀死神经元细胞导致失忆发生的。实际上，是这些产生的甾体物质影响了突触可塑性，造成了ltp和记忆形成方面的损害。

• mr morikawa」s study, results of which were published last month in the journal of neuroscience, found that repeated exposure to ethanol enhances synaptic plasticity in a key area in the brain.

  morikawa先生的研究（研究结果发表在上个月《神经科学杂志》上）表明，重复地暴露在酒精中，可以增加大脑中的一个关键部位突触的可塑性。

• to address these questions, we tested nicotinic effects on glutamatergic synaptic plasticity in vta da neurons using patch-clamp recordings in vta slice preparation.

  为阐明上述问题，我们采用膜片钳技术研究尼古丁在离体腹侧被盖区脑片上对多巴胺神经元谷氨酸能突触可塑性的影响。